29 Publications (Page 1 of 2)
2018
The mirn23a and mirn23b microrna clusters are necessary for proper hematopoietic progenitor cell production and differentiationKurkewich, J.L.⋅Boucher, A.⋅Klopfenstein, N.⋅Baskar, R.⋅Kapur, R. and Dahl, R.Experimental Hematology, vol. 59, pp. 14-29.
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2017
The miR-23a\~27a\~24-2 microRNA cluster buffers transcription and signaling pathways during hematopoiesisKurkewich, J.L.⋅Hansen, J.⋅Klopfenstein, N.⋅Zhang, H.⋅Wood, C.⋅Boucher, A.⋅Hickman, J.⋅Muench, D.E.⋅Grimes, H.L. and Dahl, R.PLoS Genetics, vol. 13.
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2016
Arid3b Is Critical for B Lymphocyte DevelopmentKurkewich, J.L.⋅Klopfenstein, N.⋅Hallas, W.M.⋅Wood, C.⋅Sattler, R.A.⋅Das, C.⋅Tucker, H.⋅Dahl, R. and Dahl, K.D.C.PLoS ONE, vol. 11.
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E2A Antagonizes PU.1 Activity through Inhibition of DNA BindingRogers, J.H.⋅Owens, K.S.⋅Kurkewich, J.⋅Klopfenstein, N.⋅Iyer, S.R.⋅Simon, M.C. and Dahl, R.BioMed Research International, vol. 2016.
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Evaluation of the mirn23a cluster through an iTRAQ-based quantitative proteomic approachLudwig, Katelyn R⋅Ludwig, K.R.⋅Dahl, Richard⋅Dahl, R.⋅Hummon, A.B. and Hummon, Amanda BJournal of Proteome Research, vol. 15, pp. 1497-1505.
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The mirn23a microRNA cluster antagonizes B cell developmentKurkewich, J.L.⋅Bikorimana, E.⋅Nguyen, T.⋅Klopfenstein, N.⋅Zhang, H.⋅Hallas, W.M.⋅Stayback, G.⋅McDowell, M.A. and Dahl, R.Journal of Leukocyte Biology, vol. 100, pp. 665-677.
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2015
MiR-24 Is Required for Hematopoietic Differentiation of Mouse Embryonic Stem CellsRoy, L.⋅Bikorimana, E.⋅Lapid, D.⋅Choi, H.⋅Nguyen, T. and Dahl, R.PLoS Genetics, vol. 11.
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2014
ARID3B increases ovarian tumor burden and is associated with a cancer stem cell gene signatureRoy, Lynn⋅Roy, Lynn⋅Samyesudhas, Serene J⋅Samyesudhas, Serene J⋅Carrasco, Martin⋅Carrasco, Martin⋅Li, Jun⋅Li, Jun⋅Joseph, Stancy⋅Joseph, Stancy⋅Dahl, Richard⋅Dahl, Richard⋅Cowden Dahl, Karen D and Dahl, Karen D. CowdenOncotarget, vol. 5, (no. 18), pp. 8366, 2014-09-30.
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Retroviral infection of murine embryonic stem cell derived embryoid body cells for analysis of hematopoietic differentiationBikorimana, E.⋅Lapid, D.⋅Choi, H. and Dahl, R.Journal of Visualized Experiments.
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2013
MiR-24 Promotes the Survival of Hematopoietic CellsNguyen, T.⋅Rich, A. and Dahl, R.PLoS ONE, vol. 8.
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2010
MIR-23A microRNA cluster inhibits B-cell developmentKong, Kimi Y⋅Kong, K.Y.⋅Owens, K.S.⋅Owens, Kristin S⋅Rogers, J.H.⋅Rogers, Jason H⋅Mullenix, J.⋅Mullenix, Jason⋅Velu, Chinavenmeni S⋅Velu, C.S.⋅Grimes, H.L.⋅Grimes, H. L⋅Dahl, R. and Dahl, RichardExperimental Hematology, vol. 38.
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2009
Development of macrophages and granulocytesDahl, R.2009
Expression of Scl in mesoderm rescues hematopoiesis in the absence of Oct-4Kong, K.Y.⋅Williamson, E.A.⋅Rogers, J.H.⋅Tran, T.⋅Hromas, R. and Dahl, R.Blood, vol. 114, pp. 60-63.
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The epidermal growth factor receptor responsive miR-125a represses mesenchymal morphology in ovarian cancer cellsCowden Dahl, K.D.⋅Dahl, R.⋅Kruichak, J.N. and Hudson, L.G.Neoplasia, vol. 11, pp. 1208-1215.
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2007
C/EBP?p30 SUMO wrestles C/EBP?p42Dahl, R.Blood, vol. 110, pp. 3089.
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Growth factor independence-1 (Gfi-1) plays a role in mediating specific granule deficiency (SGD) in a patient lacking a gene-inactivating mutation in the C/EBP? geneKhanna-Gupta, Arati⋅Khanna-Gupta, A.⋅Sun, H.⋅Sun, Hong⋅Zibello, T.⋅Zibello, Theresa⋅Han, M.L.⋅Lee, Han Myung⋅Dahl, R.⋅Dahl, Richard⋅Boxer, L.A.⋅Boxer, Laurence A⋅Berliner, N. and Berliner, NancyBlood, vol. 109, pp. 4181-4190.
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The transcriptional repressor GFI-1 antagonizes PU.1 activity through protein-protein interactionDahl, R.⋅Iyer, S.R.⋅Owens, K.S.⋅Cuylear, D.D. and Simon, M.C.Journal of Biological Chemistry, vol. 282, pp. 6473-6483.
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2006
The chemokine CXCL14 (BRAK) stimulates activated NK cell migration: Implications for the downregulation of CXCL14 in malignancyStarnes, Trevor⋅Starnes, T.⋅Rasila, Kanwaldeep K⋅Rasila, K.K.⋅Robertson, Michael J⋅Robertson, M.J.⋅Brahmi, Zacharie⋅Brahmi, Z.⋅Dahl, Richard⋅Dahl, R.⋅Christopherson, Kent⋅Christopherson, K.⋅Hromas, Robert and Hromas, R.Experimental Hematology, vol. 34, pp. 1101-1105.
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The IL-17 cytokine family members are inhibitors of human hematopoietic progenitor proliferation [1]Broxmeyer, H.E.⋅Broxmeyer, Hal E⋅Starnes, Trevor⋅Starnes, T.⋅Ramsey, H.⋅Ramsey, Heather⋅Cooper, S.⋅Cooper, Scott⋅Dahl, Richard⋅Dahl, R.⋅Williamson, E.⋅Williamson, Elizabeth⋅Hromas, R. and Hromas, RobertBlood, vol. 108, pp. 770.
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2005
Identification of a human B-cell/myeloid common progenitor by the absence of CXCR4Hou, Yong-Hao⋅Hou, Y.-H.⋅Srour, Edward F⋅Srour, E.F.⋅Ramsey, H.⋅Ramsey, Heather⋅Dahl, Richard⋅Dahl, R.⋅Broxmeyer, H.E.⋅Broxmeyer, Hal E⋅Hromas, Robert and Hromas, R.Blood, vol. 105, pp. 3488-3492.
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2003
Regulation of macrophage and neutrophil cell fates by the PU.1:C/EBP? ratio and granulocyte colony-stimulating factorDahl, R.⋅Dahl, R.⋅Walsh, J.C.⋅Walsh, JC⋅Lancki, D.⋅Lancki, D.⋅Laslo, P.⋅Laslo, P.⋅Iyer, S. R⋅Iyer, S.R.⋅Singh, Harinder⋅Singh, H.⋅Simon, Marie C and Celeste Simon, M.Nature Immunology, vol. 4, pp. 1029-1036.
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The importance of PU.1 concentration in hematopoietic lineage commitment and maturationDahl, R. and Simon, M.C.Blood Cells, Molecules, and Diseases, vol. 31, pp. 229-233.
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2002
Spi-B can functionally replace PU.1 in myeloid but not lymphoid developmentDahl, R.⋅Dahl, R.⋅Ramirez-Bergeron, D.L.⋅Ramirez Bergeron, Diana L⋅Rao, Sridhar⋅Rao, S.⋅Simon, M.C. and Simon, Marie CEMBO Journal, vol. 21, pp. 2220-2230.
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Who pays for e-junk? When faced with the mounting tide of cast-off and potentially toxic computers and other electronic products in the United States, most would agree that it is a problem in need of a solution. But as industry, government, and environmental organizations debate how best to fashion a sensible response to this new threat, one question looms large: who should pay? (Spheres of Influence)Dahl, RichardEnvironmental Health Perspectives, vol. 110, (no. 4), pp. A196, 20020401.
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2001
PU.1 exhibits partial functional redundancy with Spi-B, but not with Ets-1 or Elf-1Garrett-Sinha, L.A.⋅Garrett Sinha, Lee Ann⋅Dahl, R.⋅Dahl, R⋅Rao, S⋅Rao, S.⋅Barton, K.P.⋅Barton, K P⋅Simon, M C and Celeste Simon, M.Blood, vol. 97, pp. 2908-2912.
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